ILC3-myeloid cell crosstalk during homeostatic maintenance of the epithelial barrier and during IBD

Project Summary

It is well established that both the innate and adaptive immune systems are inappropriately activated in inflammatory bowel diseases, contributing to tissue damage. While protective innate mechanisms in intestinal epithelial cells have recently been identified, they remain less well understood. Our research aims to investigate the processes leading to IL-22–mediated epithelial protection, granuloma formation, and the granuloma-driven repair program of the epithelial barrier in the context of IBD. These studies may help us identify new innate immune targets for the treatment of IBD.

Selected project-relevant publications