Molecular mechanisms and therapeutic implications of vascular permeability defects in inflammatory bowel diseases

Project Summary

The intestinal vascular system plays a critical barrier role in inflammatory bowel diseases. Interferon-gamma (IFN-γ) is a key pathogenic factor in IBD with strong effects on the vasculature. The main goal of this project is to uncover the molecular mechanisms by which IFN-γ activation of vascular endothelial cells impairs vascular barrier functions and disrupts immune–epithelial crosstalk during the course of IBD pathogenesis. In the long term, we aim to identify novel molecular pathways involved in IFN-γ–induced dysfunction of the intestinal vascular barrier, which could serve as potential targets for future IBD therapies.

Selected project-relevant publications