Direct and indirect crosstalk of epithelial GPR15L with immune cells via the intestinal microbiome in the pathogenesis of IBD

Project Summary

While the pathogenesis of chronic inflammatory bowel diseases (IBD) is multifactorial and the intestinal microbiome is believed to play a key role, current treatments primarily target downstream immune effector mechanisms.
Modulating the microbiome to prevent early inflammatory steps appears promising but remains unestablished. The peptide G-protein-coupled receptor 15 ligand (GPR15L), secreted by colonic epithelial cells, is a ligand for the G-protein-coupled receptor 15 (GPR15) expressed on T cells migrating into the colon. We hypothesize that GPR15L influences immune interactions both directly and indirectly via microbiome-mediated mechanisms, plays a key role in IBD pathogenesis, and represents a potential therapeutic target. Our aim is to elucidate the impact of GPR15L on colitis, its underlying mechanisms, and translational potential using Gpr15l-deficient and Gpr15l-overexpressing mice, 16S rRNA sequencing, trafficking assays, human samples, and experimental interventions.

Selected project-relevant publications